The role of Ca 2+ -calmodulin stimulated protein kinase II in ischaemic stroke – A potential target for neuroprotective therapies-Reference-Cited by-同舟云学术

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The role of Ca 2+ -calmodulin stimulated protein kinase II in ischaemic stroke – A potential target for neuroprotective therapies

Published:2017-07 Issue: Volume:107 Page:33-42
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

Rostas John A.P.,Spratt Neil J.,Dickson Phillip W.,Skelding Kathryn A.

Funder

Brain Foundation

Hunter Medical Research Institute

University of Newcastle

National Health and Medical Research Council of Australia

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference71 articles.

1. Treatment of ischemic brain damage by perturbing NMDA receptor–PSD-95 protein interactions;Aarts;Science,2002

2. Small-molecule inhibitors of protein-protein interactions: progressing toward the reality;Arkin;Chem. Biol.,2014

3. Excitotoxic neuroprotection and vulnerability with CaMKII inhibition;Ashpole;Mol. Cell Neurosci.,2011

4. Calcium/Calmodulin-dependent protein kinase II (CaMKII) inhibition induces neurotoxicity via dysregulation of glutamate/calcium signaling and hyperexcitability;Ashpole;J. Biol. Chem.,2012

5. Loss of calcium/calmodulin-dependent protein kinase II activity in cortical astrocytes decreases glutamate uptake and induces neurotoxic release of ATP;Ashpole;J. Biol. Chem.,2013

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3. The GHB analogue HOCPCA improves deficits in cognition and sensorimotor function after MCAO via CaMKIIα;Journal of Cerebral Blood Flow & Metabolism;2023-04-07

4. Calcium/Calmodulin-Stimulated Protein Kinase II (CaMKII): Different Functional Outcomes from Activation, Depending on the Cellular Microenvironment;Cells;2023-01-23

5. Calcium Signalling in Heart and Vessels: Role of Calmodulin and Downstream Calmodulin-Dependent Protein Kinases;International Journal of Molecular Sciences;2022-12-17

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