A novel mechanism of non-Aβ component of Alzheimer's disease amyloid (NAC) neurotoxicity. Interplay between p53 protein and cyclin-dependent kinase 5 (Cdk5)-Reference-Cited by-同舟云学术

A novel mechanism of non-Aβ component of Alzheimer's disease amyloid (NAC) neurotoxicity. Interplay between p53 protein and cyclin-dependent kinase 5 (Cdk5)

Published:2011-02 Issue:2 Volume:58 Page:206-214
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

Kaźmierczak Anna,Czapski Grzegorz A.,Adamczyk Agata,Gajkowska Barbara,Strosznajder Joanna B.

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference83 articles.

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3. Alpha-synuclein and its neurotoxic fragment inhibit dopamine uptake into rat striatal synaptosomes. Relationship to nitric oxide;Adamczyk;Neurochem. Int.,2006

4. Alpha-synuclein potentiates Ca2+ influx through voltage-dependent Ca2+ channels;Adamczyk;Neuroreport,2006

5. Beta-amyloid stimulation of inducible nitric-oxide synthase in astrocytes is interleukin-1β- and tumor necrosis factor-a (TNFa)-dependent, and involves a TNFa receptor associated factor- and NFkappaB-inducing kinase-dependent signaling mechanism;Akama;J. Biol. Chem.,2000

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