Disturbance of mitochondrial functions caused by N-acetylglutamate and N-acetylmethionine in brain of adolescent rats: Potential relevance in aminoacylase 1 deficiency-Reference-Cited by-同舟云学术

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Disturbance of mitochondrial functions caused by N-acetylglutamate and N-acetylmethionine in brain of adolescent rats: Potential relevance in aminoacylase 1 deficiency

Published:2023-12 Issue: Volume:171 Page:105631
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

Bortoluzzi Vanessa Trindade^ORCID,Ribeiro Rafael Teixeira^ORCID,Zemniaçak Ângela Beatris^ORCID,Cunha Sâmela de Azevedo,Sass Jörn Oliver^ORCID,Castilho Roger Frigério^ORCID,Amaral Alexandre Umpierrez^ORCID,Wajner Moacir^ORCID

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference54 articles.

1. Safranine as a probe of the mitochondrial membrane potential;Åkerman;FEBS Lett.,1976

2. Isolated mild intellectual disability expands the aminoacylase 1 phenotype spectrum;Alessandrì;JIMD Rep.,2014

3. Four years follow up of ACY1 deficient patient and pedigree study;Alessandrì;Brain Dev.,2018

4. N-acetyl-l-glutamate in brain: assay, levels, and regional and subcellular distribution;Alonso;Neurochem. Res.,1991

5. Pathophysiology of maple syrup urine disease: focus on the neurotoxic role of the accumulated branched-chain amino acids and branched-chain α-keto acids;Amaral;Neurochem. Int.,2022

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