Butyrylcholinesterase: K variant, plasma activity, molecular forms and rivastigmine treatment in Alzheimer's disease in a Southern Brazilian population

Author:

Bono G.F.,Simão-Silva D.P.,Batistela M.S.,Josviak N.D.,Dias P.F.R.,Nascimento G.A.,Souza R.L.R.,Piovezan M.R.,Souza R.K.M.,Furtado-Alle L.

Funder

Coordination for Improvement of Higher Education

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference66 articles.

1. The butyrylcholinesterase K-variant shows similar cellular protein turnover and quaternary interaction to the wild type enzyme;Altamirano;J. Neurochem,2000

2. The butyrylcholinesterase K variant is a protective factor for sporadic Alzheimer's disease in women;Alvarez-Arcaya;Acta Neurol. Scand,2000

3. Changes in acetylcholinesterase and butyrylcholinesterase in Alzheimer's disease resemble embryonic development – a study of molecular forms;Arendt;Neurochem. Int,1992

4. Structure of the gene for human butyrylcholinesterase. Evidence for a single copy;Arpagaus;Biochemistry,1990

5. DNA mutation associated with the human butyrylcholinesterase K-variant and its linkage to the atypical variant mutation and other polymorphic sites;Bartels;Am. J. Hum. Genet,1992

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