Spinal cord injury causes insulin resistance associated with PI3K signaling pathway in hypothalamus-Reference-Cited by-同舟云学术

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Spinal cord injury causes insulin resistance associated with PI3K signaling pathway in hypothalamus

Published:2020-11 Issue: Volume:140 Page:104839
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

Cheng Rui-Dong,Ren Wen,Sun Peng,Tian Liang,Zhang Li,Zhang Jie,Li Jue-Bao,Ye Xiang-Ming

Funder

Natural Science Foundation of Zhejiang Province

National Natural Science Foundation of China

Scientific Research Program of Zhejiang Medical Technology

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference20 articles.

1. The effect of residual neurological deficit on oral glucose tolerance in persons with chronic spinal cord injury;Bauman;Spinal Cord,1999

2. The effect of sanggua drink extract on insulin resistance through the PI3K/AKT signaling pathway;Cai;Evid. Based Complement Alternat. Med.,2018

3. A new role for the brain in metabolic control;Carvalheira;Nat. Med.,2014

4. Perceived influence of intrinsic/extrinsic factors on participation in life activities after spinal cord injury;Cobb;Disabil. Health J.,2018

5. Spinal cord injury and type 2 diabetes: results from a population health survey;Cragg;Neurology,2013

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1. Spinal cord injury-induced metabolic impairment and steatohepatitis develops in non-obese rats and is exacerbated by premorbid obesity;Experimental Neurology;2024-09

2. Brain region changes following a spinal cord injury;Neurochemistry International;2024-03

3. A comprehensive look at the psychoneuroimmunoendocrinology of spinal cord injury and its progression: mechanisms and clinical opportunities;Military Medical Research;2023-06-09

4. Mechanism of skeletal muscle atrophy after spinal cord injury: A narrative review;Frontiers in Nutrition;2023-03-03

5. Consequences of spinal cord injury on the sympathetic nervous system;Frontiers in Cellular Neuroscience;2023-02-28

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