The Receptor for Advanced Glycation End Products (RAGE) and DIAPH1: Implications for vascular and neuroinflammatory dysfunction in disorders of the central nervous system-Reference-Cited by-同舟云学术

The Receptor for Advanced Glycation End Products (RAGE) and DIAPH1: Implications for vascular and neuroinflammatory dysfunction in disorders of the central nervous system

Published:2019-06 Issue: Volume:126 Page:154-164
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

MacLean Michael,Derk Julia,Ruiz Henry H.,Juranek Judyta K.,Ramasamy Ravichandran,Schmidt Ann Marie

Funder

United States Public Health Service

American Heart Association

JDRF

American Diabetes Association

Alzheimer's Association

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference123 articles.

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2. Immunohistochemical localization of receptor for advanced glycation end (RAGE) products in the R6/2 mouse model of Huntington's disease;Anzilotti;Brain Res. Bull.,2012

3. RAGE potentiates Abeta-induced perturbation of neuronal function in transgenic mice;Arancio;EMBO J.,2004

4. RAGE-independent autoreactive B cell activation in response to chromatin and HMGB1/DNA immune complexes;Avalos;Autoimmunity,2010

5. The possible involvement of HLA class III haplotype (RAGE, HSP70 and TNF genes) in alzheimer's disease;Boiocchi;Curr. Alzheimer Res.,2015

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