Defective cell proliferation is an attribute of overexpressed Notch1 receptor and impaired autophagy in Fanconi Anemia-Reference-Cited by-同舟云学术

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Defective cell proliferation is an attribute of overexpressed Notch1 receptor and impaired autophagy in Fanconi Anemia

Published:2020-11 Issue:6 Volume:112 Page:4628-4639
ISSN:0888-7543
Container-title:Genomics
language:en
Short-container-title:Genomics

Author:

Zipporah E Binita,Patra Bamadeb,Govarthanan Kavitha,Yadav Rajesh,Mohan Sheila,Shyamsunder Pavithra,Verma Rama Shanker

Publisher

Elsevier BV

Subject

Genetics

Reference51 articles.

1. Fanconi anemia and its diagnosis;Auerbach;Mutat. Res. Fundam. Mol. Mech. Mutagen.,2009

2. The Fanconi anaemia/BRCA pathway;D’Andrea;Nat. Rev. Cancer,2003

3. Ubiquitylation and the Fanconi anemia pathway;Garner;FEBS Lett.,2011

4. Oxidative stress in Fanconi anaemia: from cells and molecules towards prospects in clinical management;Pagano;Biol. Chem.,2012

5. Identification of novel target genes involved in Indian Fanconi anemia patients using microarray;Shyamsunder;Gene,2013

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1. Identification of a Novel Curcumin Derivative Influencing Notch Pathway and DNA Damage as a Potential Therapeutic Agent in T-ALL;Cancers;2022-11-24

2. Label-free serum albumin nanoparticles for bioimaging and Trojan horse-like drug delivery;Journal of Science: Advanced Materials and Devices;2022-03

3. Targeting Notch to Maximize Chemotherapeutic Benefits: Rationale, Advanced Strategies, and Future Perspectives;Cancers;2021-10-12

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