Translocator Protein (18 kDa) Downregulation Contributes to Low Cortisol and Neurosteroid Levels in Patients with Post Traumatic Stress Disorder-Reference-Cited by-同舟云学术

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Translocator Protein (18 kDa) Downregulation Contributes to Low Cortisol and Neurosteroid Levels in Patients with Post Traumatic Stress Disorder

Published:2017-07 Issue: Volume:104 Page:72
ISSN:0306-9877
Container-title:Medical Hypotheses
language:en
Short-container-title:Medical Hypotheses

Author:

Bechard Brendan

Publisher

Elsevier BV

Subject

General Medicine

Reference11 articles.

1. Et al. Hypothalamic-pituitary-adrenal alterations in PTSD Expanding the stress-response spectrum;Yehuda,1995

2. Hypothalamic-pituitary-adrenal dysfunction in post-traumatic stress disorder;Yehuda;Biol Psychiatry,1991

3. LOW urinary cortisol excretion in Holocaust survivors with post-traumatic stress disorder;Yehuda;Am J Psychiatry,1995

4. Increased lymphocyte glucocorticoid receptor number in post-traumatic stress disorder;Yehuda;Am J Psychiatry,1991

5. Enhanced suppression of cortisol following dexamethasone administration in combat veterans with post-traumatic stress disorder and major depressive disorder;Yehuda;Am J Psychiatry,1993

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