Tgif1 and SnoN modified chondrocytes or stem cells for tendon–bone insertion regeneration
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference45 articles.
1. The skeletal attachment of tendons–tendon “entheses”;Benjamin;Comp Biochem Physiol A Mol Integr Physiol,2002
2. Where tendons and ligaments meet bone: attachment sites (‘entheses’) in relation to exercise and/or mechanical load;Benjamin;J Anat,2006
3. Transforming growth factors beta coordinate cartilage and tendon differentiation in the developing limb mesenchyme;Lorda-Diez;J Biol Chem,2009
4. SnoN suppresses maturation of chondrocytes by mediating signal cross-talk between transforming growth factor-beta and bone morphogenetic protein pathways;Kawamura;J Biol Chem,2012
5. Delayed repair of tendon to bone injuries leads to decreased biomechanical properties and bone loss;Galatz;J Orthop Res,2005
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. The tissues and regulatory pattern of limb chondrogenesis;Developmental Biology;2020-07
2. Transcriptional cofactors Ski and SnoN are major regulators of the TGF-β/Smad signaling pathway in health and disease;Signal Transduction and Targeted Therapy;2018-06-08
3. Silencing of TGIF1 in bone mesenchymal stem cells applied to the post-operative rotator cuff improves both functional and histologic outcomes;Journal of Molecular Histology;2015-03-18
4. TGIF1 Gene Silencing in Tendon-Derived Stem Cells Improves the Tendon-to-Bone Insertion Site Regeneration;Cellular Physiology and Biochemistry;2015
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