ε-Glycation, APP and Aβ in ageing and Alzheimer disease: A hypothesis-Reference-Cited by-同舟云学术

ε-Glycation, APP and Aβ in ageing and Alzheimer disease: A hypothesis

Published:2006-01 Issue:5 Volume:66 Page:898-906
ISSN:0306-9877
Container-title:Medical Hypotheses
language:en
Short-container-title:Medical Hypotheses

Author:

Schmitt H. Peter

Publisher

Elsevier BV

Subject

General Medicine

Reference142 articles.

1. Inhibition of advanced glycation endproduct formation by acetaldehyde: role in the cardioprotective effect of ethanol;Al-Abed;Proc Natl Acad Sci USA,1999

2. RAGE potentiates Aβ-induced perturbation of neuronal function in transgenic mice;Arancio;EMBO J,2004

3. Oxidative degradation of protein-bound Amadori products: formation of Nε-carboxymethyllysine and carboxymethyl amino acids as indicators of the extent of non-enzymatic glycosylation;Badoud,1990

4. Key factors in Alzheimer’s disease: β-amyloid precursor protein processing, metabolism and intraneuronal transport;Bayer;Brain Pathol,2001

5. Amyloid precursor protein (APP) and βA4 amyloid in the etiology of Alzheimer’s disease: precursor-product relationships in the derangement of neuronal function;Beyreuther;Brain Pathol,1991

Cited by 19 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The status of glycation in protein aggregation;International Journal of Biological Macromolecules;2017-07

2. Alzheimer's Disease: From Animal Models to the Human Syndrome;Update on Dementia;2016-09-28

3. Protective action of green tea catechins in neuronal mitochondria during aging;Frontiers in Bioscience;2015

4. Protein Folding: From Normal Cellular Function to Pathophysiology;Proteostasis and Chaperone Surveillance;2015

5. Biology of aging brain;Indian Journal of Pathology and Microbiology;2010