P.583 Polymorphisms in BDNF, AKT1, GSK3B genes: possible association with antipsychotic-induced hyperprolactinemia in schizophrenia patients-Reference-Cited by-同舟云学术

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P.583 Polymorphisms in BDNF, AKT1, GSK3B genes: possible association with antipsychotic-induced hyperprolactinemia in schizophrenia patients

Published:2020-11 Issue: Volume:40 Page:S331-S332
ISSN:0924-977X
Container-title:European Neuropsychopharmacology
language:en
Short-container-title:European Neuropsychopharmacology

Author:

Ivanova S.A.,Paderina D.Z.,Boiko A.S.,Pozhidaev I.V.,Kornetova E.G.,Wilffert B.,Loonen A.J.M.

Funder

Russian Foundation for Basic Research

Publisher

Elsevier BV

Subject

Pharmacology (medical),Biological Psychiatry,Psychiatry and Mental health,Clinical Neurology,Neurology,Pharmacology

Reference5 articles.

1. A pharmacogenetic study of patients with schizophrenia from West Siberia gets insight into dopaminergic mechanisms of antipsychotic-induced hyperprolactinemia;Osmanova;BMC Medical Genetics,2019

2. Identification of 5-hydroxytryptamine receptor gene polymorphisms modulating hyperprolactinaemia in antipsychotic drug-treated patients with schizophrenia;Ivanova;The World Journal of Biological Psychiatry,2017

3. Association between eight P-glycoprotein (MDR1/ABCB1) gene polymorphisms and antipsychotic drug-induced hyperprolactinemia;Geers;British Journal of Clinical Pharmacology.,2020

4. The functional variant rs334558 of GSK3B is associated with remission in patients with depressive disorders;Levchenko;Pharmacogenomics and Personalized Medicine Volume,2018

5. No evidence so far of a major role of AKT1 and GSK3B in the pathogenesis of antipsychotic-induced tardive dyskinesia;Levchenko;Human Psychopharmacology: Clinical and Experimental,2019

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