Involvement of IL-1β in acute stress-induced worsening of cerebral ischaemia in rats-Reference-Cited by-同舟云学术

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Involvement of IL-1β in acute stress-induced worsening of cerebral ischaemia in rats

Published:2007-09 Issue:9 Volume:17 Page:600-607
ISSN:0924-977X
Container-title:European Neuropsychopharmacology
language:en
Short-container-title:European Neuropsychopharmacology

Author:

Caso Javier R.,Moro María A.,Lorenzo Pedro,Lizasoain Ignacio,Leza Juan C.

Publisher

Elsevier BV

Subject

Pharmacology (medical),Biological Psychiatry,Psychiatry and Mental health,Clinical Neurology,Neurology,Pharmacology

Reference53 articles.

1. Beta-amyloid stimulation of inducible nitric-oxide synthase in astrocytes is interleukin-1beta- and tumor necrosis factor-alpha (TNFalpha)-dependent, and involves a TNFalpha receptor-associated factor- and NFkappaB-inducing kinase-dependent signaling mechanism;Akama;J. Biol. Chem.,2000

2. Interleukin-1 and neuronal injury;Allan;Nat. Rev., Immunol.,2005

3. Cytokines, stress, and depressive illness;Anisman;Brain Behav. Immun.,2002

4. Corticotropin-releasing factor-producing neurons in the rat activated by interleukin-1;Berkenbosch;Science,1987

5. Immunoregulatory feedback between interleukin-1 and glucocorticoid hormones;Besedovsky;Science,1986

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