Endoplasmic reticulum-translocation is essential for APOL1 cellular toxicity-Reference-Cited by-同舟云学术

Endoplasmic reticulum-translocation is essential for APOL1 cellular toxicity

Published:2022-01 Issue:1 Volume:25 Page:103717
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Kruzel-Davila Etty,Bavli-Kertselli Ira,Ofir Ayala,Cheatham Amber M.,Shemer Revital,Zaknoun Eid,Chornyy Sergiy,Tabachnikov Orly,Davis Shamara E.,Khatua Atanu K.,Skorecki Karl,Popik Waldemar

Funder

Israel Science Foundation

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference44 articles.

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4. APOL1-G0 or APOL1-G2 transgenic models develop preeclampsia but not kidney disease;Bruggeman;J. Am. Soc. Nephrol.,2016

5. Identifying the Intracellular function of APOL1;Bruggeman;J. Am. Soc. Nephrol.,2017

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1. The Two Levels of Podocyte Dysfunctions Induced by Apolipoprotein L1 Risk Variants;Kidney and Dialysis;2024-06-07

2. APOL1 nephropathy – a population genetics success story;Current Opinion in Nephrology & Hypertension;2024-02-28

3. Apolipoprotein L1 (APOL1) renal risk variant-mediated podocyte cytotoxicity depends on African haplotype and surface expression;Scientific Reports;2024-02-14

4. A SNARE protective pool antagonizes APOL1 renal toxicity in Drosophila nephrocytes;Cell & Bioscience;2023-11-04

5. Novel Therapies in APOL1-Mediated Kidney Disease: From Molecular Pathways to Therapeutic Options;Kidney International Reports;2023-11