Anoxia-induced hippocampal LTP is regeneratively produced by glutamate and nitric oxide from the neuro-glial-endothelial axis-Reference-Cited by-同舟云学术

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Anoxia-induced hippocampal LTP is regeneratively produced by glutamate and nitric oxide from the neuro-glial-endothelial axis

Published:2024-04 Issue:4 Volume:27 Page:109515
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Wang Han-Ying,Takagi Hiroshi,Stoney Patrick N.,Echeverria Anai,Kuhn Bernd,Hsu Kuei-Sen,Takahashi Tomoyuki^ORCID

Funder

Okinawa Institute of Science and Technology

Publisher

Elsevier BV

Reference58 articles.

1. A reversible type of neuronal injury following ischemia in the gerbil hippocampus;Kirino;Stroke,1986

2. Human amnesia and the medial temporal region: enduring memory impairment following a bilateral lesion limited to field CA1 of the hippocampus;Zola-Morgan;J. Neurosci.,1986

3. Glutamate neurotoxicity and diseases of the nervous system;Choi;Neuron,1988

4. A selective LTP of NMDA receptor-mediated currents induced by anoxia in CA1 hippocampal neurons;Crépel;J. Neurophysiol.,1993

5. Long-term potentiation in hippocampal slices induced by temporary suppression of glycolysis;Tekkök;J. Neurophysiol.,1995

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The Unexpected Role of the Endothelial Nitric Oxide Synthase at the Neurovascular Unit: Beyond the Regulation of Cerebral Blood Flow;International Journal of Molecular Sciences;2024-08-21

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