TRPV6 is a potential regulator of bone resorption in bone loss induced by estrogen deficiency-Reference-Cited by-同舟云学术

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TRPV6 is a potential regulator of bone resorption in bone loss induced by estrogen deficiency

Published:2021-11 Issue:11 Volume:24 Page:103261
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Ma Jun,Lu Jiajia,Zhou Zhibin,Lu Nan,He Jia,Zhu Lei,Ye Tianwen

Funder

Shanghai Jiao Tong University

Second Military Medical University

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference38 articles.

1. 1,25-Dihydroxyvitamin D3/VDR-mediated induction of FGF23 as well as transcriptional control of other bone anabolic and catabolic genes that orchestrate the regulation of phosphate and calcium mineral metabolism;Barthel;J. Steroid Biochem. Mol. Biol.,2007

2. Expression of mRNA for the alpha 1 subunit of voltage-gated calcium channels in human osteoblast-like cell lines and normal human osteoblasts;Bary;Calcif Tissue Int.,2000

3. Steroid hormone receptors: many actors in search of a plot;Beato;Cell,1995

4. TRPV6 and Ca1.3 mediate distal small intestine calcium absorption before weaning;Beggs;Cell Mol. Gastroenterol. Hepatol.,2019

5. Transient versus sustained phosphorylation and nuclear accumulation of ERKs underlie anti- versus pro-apoptotic effects of estrogens;Chen;J. Biol.Chem.,2005

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1. Fibroblast growth factor-21 potentiates the stimulatory effects of 1,25-dihydroxyvitamin D3 on transepithelial calcium transport and TRPV6 Ca2+ channel expression;Biochemical and Biophysical Research Communications;2024-11

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4. Integration of single‐cell and RNA‐seq data to explore the role of focal adhesion‐related genes in osteoporosis;Journal of Cellular and Molecular Medicine;2024-03-27

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