TACI Constrains TH17 Pathogenicity and Protects against Gut Inflammation
Author:
Funder
Agency for Science, Technology and Research
Publisher
Elsevier BV
Subject
Multidisciplinary
Reference44 articles.
1. TACI deficiency leads to alternatively activated macrophage phenotype and susceptibility to Leishmania infection;Allman;Proc. Natl. Acad. Sci. U S A,2015
2. The costimulatory molecule ICOS regulates the expression of c-Maf and IL-21 in the development of follicular T helper cells and TH -17 cells;Bauquet;Nat. Immunol.,2009
3. IRF4 and its regulators: evolving insights into the pathogenesis of inflammatory arthritis?;Biswas;Immunol. Rev.,2010
4. TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts;Bossen;Blood,2008
5. The development of inflammatory TH-17 cells requires interferon-regulatory factor 4;Brüstle;Nat. Immunol.,2007
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