PABPC1 mediates degradation of C9orf72-FTLD/ALS GGGGCC repeat RNA-Reference-Cited by-同舟云学术

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PABPC1 mediates degradation of C9orf72-FTLD/ALS GGGGCC repeat RNA

Published:2024-03 Issue:3 Volume:27 Page:109303
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Uozumi Ryota,Mori Kohji^ORCID,Gotoh Shiho,Miyamoto Tesshin,Kondo Shizuko,Yamashita Tomoko,Kawabe Yuya,Tagami Shinji,Akamine Shoshin,Ikeda Manabu

Funder

Japan Science and Technology Agency Fusion Oriented REsearch for disruptive Science and Technology

Japan Society for the Promotion of Science

Takeda Science Foundation

AMED

Osaka University

SENSHIN Medical Research Foundation

Japan Science and Technology Agency

Publisher

Elsevier BV

Reference62 articles.

1. Pathogenic determinants and mechanisms of ALS/FTD linked to hexanucleotide repeat expansions in the C9orf72 gene;Wen;Neurosci. Lett.,2017

2. Frontotemporal Lobar Degeneration (FTLD): Review and Update for Clinical Neurologists;Hernandez;Curr. Alzheimer Res.,2018

3. Genotype-phenotype links in frontotemporal lobar degeneration;Van Mossevelde;Nat. Rev. Neurol.,2018

4. Unconventional Translation of C9ORF72 GGGGCC Expansion Generates Insoluble Polypeptides Specific to c9FTD/ALS;Ash;Neuron,2013

5. The C9orf72 GGGGCC repeat is translated into aggregating dipeptide-repeat proteins in FTLD/ALS;Mori;Science,2013

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