PRMT1 Deficiency in Mouse Juvenile Heart Induces Dilated Cardiomyopathy and Reveals Cryptic Alternative Splicing Products-Reference-Cited by-同舟云学术

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PRMT1 Deficiency in Mouse Juvenile Heart Induces Dilated Cardiomyopathy and Reveals Cryptic Alternative Splicing Products

Published:2018-10 Issue: Volume:8 Page:200-213
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Murata Kazuya,Lu Weizhe,Hashimoto Misuzu,Ono Natsumi,Muratani Masafumi,Nishikata Kana,Kim Jun-Dal,Ebihara Shizufumi,Ishida Junji,Fukamizu Akiyoshi

Funder

Uehara Memorial Foundation

Japan Society for the Promotion of Science (JSPS)

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference74 articles.

1. Protein interaction screening for the ankyrin repeats and suppressor of cytokine signaling (SOCS) box (ASB) family identify Asb11 as a novel endoplasmic reticulum resident ubiquitin ligase;Andresen;J. Biol. Chem.,2014

2. The Caenorhabditis elegans unc-49 locus encodes multiple subunits of a heteromultimeric GABA receptor;Bamber;J. Neurosci.,1999

3. Roles of nebulin family members in the heart;Bang;Circ. J.,2015

4. Alternative splicing as a regulator of development and tissue identity;Baralle;Nat. Rev. Mol. Cell Biol.,2017

5. A mutation in the glutamate-rich region of RNA-binding motif protein 20 causes dilated cardiomyopathy through missplicing of titin and impaired Frank-Starling mechanism;Beqqali;Cardiovasc. Res.,2016

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5. PRMT1 in human neoplasm: cancer biology and potential therapeutic target;Cell Communication and Signaling;2024-02-08

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