Dysregulated innate immune signaling cooperates with RUNX1 mutations to transform an MDS-like disease to AML-Reference-Cited by-同舟云学术

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Dysregulated innate immune signaling cooperates with RUNX1 mutations to transform an MDS-like disease to AML

Published:2024-06 Issue:6 Volume:27 Page:109809
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Barreyro Laura,Sampson Avery M.,Hueneman Kathleen,Choi Kwangmin,Christie Susanne,Ramesh Vighnesh,Wyder Michael,Wang Dehua,Pujato Mario,Greis Kenneth D.,Huang Gang,Starczynowski Daniel T.

Funder

Cincinnati Children’s Research Foundation

Leukemia and Lymphoma Society

National Institute of Diabetes and Digestive and Kidney Diseases

CancerFree KIDS

Mark Foundation For Cancer Research

Cincinnati Children's Hospital Medical Center

National Institutes of Health

Publisher

Elsevier BV

Reference64 articles.

1. Genetics of progression from MDS to secondary leukemia;Menssen;Blood,2020

2. The Genetic Landscape of Myelodysplastic Neoplasm Progression to Acute Myeloid Leukemia;Bănescu;Int. J. Mol. Sci.,2023

3. Innate immune pathways and inflammation in hematopoietic aging, clonal hematopoiesis, and MDS;Trowbridge;J. Exp. Med.,2021

4. Chronic immune response dysregulation in MDS pathogenesis;Barreyro;Blood,2018

5. The central role of inflammatory signaling in the pathogenesis of myelodysplastic syndromes;Sallman;Blood,2019

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