Differential response of C9orf72 transcripts following neuronal depolarization
Author:
Funder
NIH
Publisher
Elsevier BV
Subject
Multidisciplinary
Reference69 articles.
1. A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD;Renton;Neuron,2011
2. Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS;DeJesus-Hernandez;Neuron,2011
3. hnRNP A3 binds to GGGGCC repeats and is a constituent of p62-positive/TDP43-negative inclusions in the hippocampus of patients with C9orf72 mutations;Mori;Acta Neuropathol.,2013
4. RNA toxicity from the ALS/FTD C9ORF72 expansion is mitigated by antisense intervention;Donnelly;Neuron,2013
5. C9orf72 nucleotide repeat structures initiate molecular cascades of disease;Haeusler;Nature,2014
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