Recurrent der(9;18) in essential thrombocythemia with JAK2 V617F is highly linked to myelofibrosis development
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Genetics,Molecular Biology
Reference21 articles.
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2. Cancer Genome Project. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders;Baxter;Lancet,2005
3. A gain-of-function mutation of JAK2 in myeloproliferative disorders;Kralovics;N Engl J Med,2005
4. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis;Levine;Cancer Cell,2005
5. Identification of an acquired JAK2 mutation in polycythemia vera;Zhao;J Biol Chem.,2005
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. ESSENTIAL THROMBOCYTHAEMIA : A CASE REPORT;Journal of Evolution of Medical and Dental Sciences;2015-04-09
2. Translocation t(1;9) is a recurrent cytogenetic abnormality associated with progression of essential thrombocythemia patients displaying the JAK2 V617F mutation;Leukemia Research;2011-09
3. der(1)t(1;19)(p13;p13.1) in two elderly patients with myeloid neoplasms: New case reports and review of the literature;Leukemia Research;2009-08
4. Pathological interactions between hematopoietic stem cells and their niche revealed by mouse models of primary myelofibrosis;Expert Review of Hematology;2009-06
5. Derivative (1)t(1;19)(p13;p13) in the setting of myelofibrosis with JAK2 V617F;Cancer Genetics and Cytogenetics;2009-06
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