Polycythemia vera transforming to acute myeloid leukemia and complex abnormalities including 9p homogeneously staining region with amplification of MLLT3, JMJD2C, JAK2, and SMARCA2-Reference-Cited by-同舟云学术

Polycythemia vera transforming to acute myeloid leukemia and complex abnormalities including 9p homogeneously staining region with amplification of MLLT3, JMJD2C, JAK2, and SMARCA2

Published:2008-01 Issue:1 Volume:180 Page:51-55
ISSN:0165-4608
Container-title:Cancer Genetics and Cytogenetics
language:en
Short-container-title:Cancer Genetics and Cytogenetics

Author:

Hélias Catherine,Struski Stephanie,Gervais Carine,Leymarie Vincent,Mauvieux Laurent,Herbrecht Raoul,Lessard Michel

Publisher

Elsevier BV

Subject

Cancer Research,Genetics,Molecular Biology

Reference24 articles.

1. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders;Baxter;Lancet,2005

2. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera;James;Nature,2005

3. A gain-of-function mutation of JAK2 in myeloproliferative disorders;Kralovics;N Engl J Med,2005

4. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis;Levine;Cancer Cell,2005

5. Gains on 9p are common genomic aberrations in idiopathic myelofibrosis: a comparative genomic hybridization study;Al-Assar;Br J Haematol,2005

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