Endothelin-1 mediated high glucose-induced epithelial–mesenchymal transition in renal tubular cells-Reference-Cited by-同舟云学术

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Endothelin-1 mediated high glucose-induced epithelial–mesenchymal transition in renal tubular cells

Published:2014-04 Issue:1 Volume:104 Page:176-182
ISSN:0168-8227
Container-title:Diabetes Research and Clinical Practice
language:en
Short-container-title:Diabetes Research and Clinical Practice

Author:

Tang Lin,Li Hui,Gou Rong,Cheng Genyang,Guo Yuanyuan,Fang Yudong,Chen Fengmei

Funder

Key Disciplines Laboratory of Clinical Medicine of Henan Province

Publisher

Elsevier BV

Subject

Endocrinology,General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference37 articles.

1. Epithelial–mesenchymal transition in renal tubular cells in the pathogenesis of progressive tubulo-interstitial fibrosis;Mucsi;Acta Physiol Hung,2007

2. Role for integrin-linked kinase in mediating tubular epithelial to mesenchymal transition and renal interstitial fibrogenesis;Li;J Clin Invest,2003

3. Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE);Oldfield;J Clin Invest,2001

4. Endothelin-1 induces alveolar epithelial–mesenchymal transition through endothelin type A receptor-mediated production of TGF-beta1;Jain;Am J Respir Cell Mol Biol,2007

5. Endothelin-1 is required during epithelial to mesenchymal transition in ovarian cancer progression;Rosano;Exp Biol Med (Maywood),2006

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