Muscle specific kinase (MuSK) activation preserves neuromuscular junctions in the diaphragm but is not sufficient to provide a functional benefit in the SOD1G93A mouse model of ALS-Reference-Cited by-同舟云学术

Muscle specific kinase (MuSK) activation preserves neuromuscular junctions in the diaphragm but is not sufficient to provide a functional benefit in the SOD1G93A mouse model of ALS

Published:2019-04 Issue: Volume:124 Page:340-352
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Sengupta-Ghosh Arundhati,Dominguez Sara L.,Xie Luke,Barck Kai H.,Jiang Zhiyu,Earr Timothy,Imperio Jose,Phu Lilian,Budayeva Hanna G.,Kirkpatrick Donald S.,Cai Hao,Eastham-Anderson Jeffrey,Ngu Hai,Foreman Oded,Hedehus Maj,Reichelt Michael,Hotzel Isidro,Shang Yonglei,Carano Richard A.D.,Ayalon Gai,Easton Amy

Publisher

Elsevier BV

Subject

Neurology

Reference54 articles.

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5. Vacuolization correlates with spin-spin relaxation time in motor brainstem nuclei and behavioural tests in the transgenic G93A-SOD1 mouse model of ALS

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