Communication defects with astroglia contribute to early impairments in the motor cortex plasticity of SOD1G93A mice-Reference-Cited by-同舟云学术

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Communication defects with astroglia contribute to early impairments in the motor cortex plasticity of SOD1G93A mice

Published:2024-04 Issue: Volume:193 Page:106435
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Costa-Pinto Sara,Gonçalves-Ribeiro Joana,Tedim-Moreira Joana,Socodato Renato,Relvas João B.,Sebastião Ana M.,Vaz Sandra H.

Funder

Horizon 2020 Framework Programme

Foundation for Science and Technology

International Society of Nephrology

Bureau of International Security and Nonproliferation

Ministério da Ciência, Tecnologia e Ensino Superior

International Society for Neurochemistry

Publisher

Elsevier BV

Reference81 articles.

1. Elevated cortical extracellular fluid glutamate in transgenic mice expressing human mutant (G93A) Cu/Zn superoxide dismutase;Alexander;J. Neurochem.,2000

2. Capabilities of the WinLTP data acquisition program extending beyond basic LTP experimental functions;Anderson;J. Neurosci. Methods,2007

3. Plasma glutamate and glycine levels in patients with amyotrophic lateral sclerosis: the effect of riluzole treatment;Andreadou;Clin. Neurol. Neurosurg.,2008

4. Gliotransmitters travel in time and space;Araque;Neuron,2014

5. Going the extra (synaptic) mile: excitotoxicity as the road toward neurodegenerative diseases;Armada-Moreira;Front. Cell. Neurosci.,2020

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1. Modulation of Astroglial Glutamate Transporters by CB1 Receptor Shapes Endocannabinoid-Mediated Long-Term Depression in the Medial Prefrontal Cortex;2024-04-19

2. Increasing the vitamin C transporter SVCT2 in microglia improves synaptic plasticity and restrains memory impairments in Alzheimer’s disease models;2024-03-04

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