Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes-Reference-Cited by-同舟云学术

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Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

Published:2017-10 Issue: Volume:106 Page:76-88
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

García-Cerro Susana,Rueda Noemí,Vidal Verónica,Lantigua Sara,Martínez-Cué Carmen

Funder

Jerome Lejeune Foundation

Fundación Tatiana Pérez de Guzmán el Bueno

Ministry of Economy and Competitiveness

Publisher

Elsevier BV

Subject

Neurology

Reference112 articles.

1. DYRK1A in neurodegeneration and cancer: molecular basis and clinical implications;Abbassi;Pharmacol. Ther.,2015

2. DYRK1A BAC transgenic mice show altered synaptic plasticity with learning and memory defects;Ahn;Neurobiol. Dis.,2006

3. Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments;Alonso;Proc. Natl. Acad. Sci. U. S. A.,2001

4. Alzheimer's disease hyperphosphorylated tau sequesters normal tau into tangles of filaments and disassembles microtubules;Alonso;Nat. Med.,1996

5. Role of abnormally phosphorylated tau in the breakdown of microtubules in Alzheimer disease;Alonso;Proc. Natl. Acad. Sci. U. S. A.,1994

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