Timed conditional null of connexin26 in mice reveals temporary requirements of connexin26 in key cochlear developmental events before the onset of hearing-Reference-Cited by-同舟云学术

Timed conditional null of connexin26 in mice reveals temporary requirements of connexin26 in key cochlear developmental events before the onset of hearing

Published:2015-01 Issue: Volume:73 Page:418-427
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Chang Qing,Tang Wenxue,Kim Yeunjung,Lin Xi

Funder

National Institute on Deafness and Other Communication Disorders

Publisher

Elsevier BV

Subject

Neurology

Reference38 articles.

1. Connexins 26 and 30 are co-assembled to form gap junctions in the cochlea of mice;Ahmad;Biochem. Biophys. Res. Commun.,2003

2. Restoration of connexin26 protein level in the cochlea completely rescues hearing in a mouse model of human connexin30-linked deafness;Ahmad;Proc. Natl. Acad. Sci. U. S. A.,2007

3. Impaired permeability to Ins(1,4,5)P3 in a mutant connexin underlies recessive hereditary deafness;Beltramello;Nat. Cell Biol.,2005

4. Quo vadis, hair cell regeneration?;Brigande;Nat. Neurosci.,2009

5. Gap junction mediated intercellular metabolite transfer in the cochlea is compromised in connexin30 null mice;Chang;PLoS ONE,2008

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