Maintaining energy homeostasis is an essential component of WldS-mediated axon protection-Reference-Cited by-同舟云学术

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Maintaining energy homeostasis is an essential component of WldS-mediated axon protection

Published:2013-11 Issue: Volume:59 Page:69-79
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Shen Hua,Hyrc Krzysztof L.,Goldberg Mark P.

Funder

National Institute of Health (NIH) Neuroscience Blueprint Interdisciplinary Center Core Grant

NIH National Center for Advancing Translational Sciences Grant

NIH Grants

Publisher

Elsevier BV

Subject

Neurology

Reference61 articles.

1. The slow Wallerian degeneration gene in vivo protects motor axons but not their cell bodies after avulsion and neonatal axotomy;Adalbert;Eur. J. Neurosci.,2006

2. Acute energy restriction triggers Wallerian degeneration in mouse;Alvarez;Exp. Neurol.,2008

3. Increased nuclear NAD biosynthesis and SIRT1 activation prevent axonal degeneration;Araki;Science,2004

4. WldS requires Nmnat1 enzymatic activity and N16-VCP interactions to suppress Wallerian degeneration;Avery;J. Cell Biol.,2009

5. Wld(s) prevents axon degeneration through increased mitochondrial flux and enhanced mitochondrial ca(2+) buffering;Avery;Curr. Biol.,2012

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1. NAD⁺, Axonal Maintenance, and Neurological Disease;Antioxidants & Redox Signaling;2023-12-01

2. Effect of Exercise and Oral Niacinamide Mononucleotide on Improving Mitochondrial Autophagy in Alzheimer’s Disease;Nutrients;2023-06-23

3. Pivotal roles for membrane phospholipids in axonal degeneration;The International Journal of Biochemistry & Cell Biology;2022-09

4. Programming axonal mitochondrial maintenance and bioenergetics in neurodegeneration and regeneration;Neuron;2022-06

5. NAD+-dependent mechanism of pathological axon degeneration;Cell Insight;2022-04

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