APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizures-Reference-Cited by-同舟云学术

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APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizures

Published:2017-02 Issue: Volume:98 Page:149-157
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Pirone Antonella,Alexander Jonathan,Lau Lauren A.,Hampton David,Zayachkivsky Andrew,Yee Amy,Yee Audrey,Jacob Michele H.,Dulla Chris G.

Funder

Citizens United for Research in Epilepsy (CURE) Infantile Spasms Initiative

NINDS

NIH NIDCD

Synapse Neurobiology Training Grant

Tufts Center for Neuroscience Research

PI DOD

Publisher

Elsevier BV

Subject

Neurology

Reference58 articles.

1. Gabapentin attenuates hyperexcitability in the freeze-lesion model of developmental cortical malformation;Andresen;Neurobiol. Dis.,2014

2. Molecular characterization of a cohort of 73 patients with infantile spasms syndrome;Boutry-Kryza;Eur. J. Med. Genet.,2015

3. Increased seizure susceptibility and cortical malformation in beta-catenin mutant mice;Campos;Biochem. Biophys. Res. Commun.,2004

4. Astrocyte control of synaptic NMDA receptors contributes to the progressive development of temporal lobe epilepsy;Clasadonte;Proc. Natl. Acad. Sci. U. S. A.,2013

5. Corpus callosum agenesis, severe mental retardation, epilepsy, and dyskinetic quadriparesis due to a novel mutation in the homeodomain of ARX;Conti;Am. J. Med. Genet. A,2011

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