LANP mediates neuritic pathology in Spinocerebellar ataxia type 1-Reference-Cited by-同舟云学术

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LANP mediates neuritic pathology in Spinocerebellar ataxia type 1

Published:2012-12 Issue:3 Volume:48 Page:526-532
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Cvetanovic Marija,Kular Rupinder K.,Opal Puneet

Funder

U.S. National Institutes of Health

National Organization for Rare Disorders

National Ataxia Foundation

Brain Research Foundation

Publisher

Elsevier BV

Subject

Neurology

Reference45 articles.

1. Aberrant interactions of transcriptional repressor proteins with the Huntington's disease gene product, huntingtin;Boutell;Hum. Mol. Genet.,1999

2. SCA1 transgenic mice: a model for neurodegeneration caused by an expanded CAG trinucleotide repeat;Burright;Cell,1995

3. Vascular endothelial growth factor ameliorates the ataxic phenotype in a mouse model of spinocerebellar ataxia type 1;Cvetanovic;Nat. Med.,2011

4. The role of LANP and ataxin 1 in E4F-mediated transcriptional repression;Cvetanovic;EMBO Rep.,2007

5. The amino terminus of the human AR is target for corepressor action and antihormone agonism;Dotzlaw;Mol. Endocrinol.,2002

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1. Longitudinal single-cell transcriptional dynamics throughout neurodegeneration in SCA1;Neuron;2023-11

2. A survey of protein interactions and posttranslational modifications that influence the polyglutamine diseases;Frontiers in Molecular Neuroscience;2022-09-14

3. Developmental Alterations in Adult‐Onset Neurodegenerative Disorders: Lessons from Polyglutamine Diseases;Movement Disorders;2021-05-20

4. Gene Deregulation and Underlying Mechanisms in Spinocerebellar Ataxias With Polyglutamine Expansion;Frontiers in Neuroscience;2020-06-09

5. Pathogenic mechanisms underlying spinocerebellar ataxia type 1;Cellular and Molecular Life Sciences;2020-04-18

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