Gene expression profiling highlights defective myogenesis in DMD patients and a possible role for bone morphogenetic protein 4-Reference-Cited by-同舟云学术

Gene expression profiling highlights defective myogenesis in DMD patients and a possible role for bone morphogenetic protein 4

Published:2006-07 Issue:1 Volume:23 Page:228-236
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Sterrenburg Ellen,van der Wees Caroline G.C.,White Stefan J.,Turk Rolf,de Menezes Renée X.,van Ommen Gert-Jan B.,den Dunnen Johan T.,'t Hoen Peter A.C.

Publisher

Elsevier BV

Subject

Neurology

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2. Therapeutic antisense-induced exon skipping in cultured muscle cells from six different DMD patients;Aartsma-Rus;Hum. Mol. Genet.,2003

3. Deflazacort but not prednisone improves both muscle repair and fiber growth in diaphragm and limb muscle in vivo in the mdx dystrophic mouse;Anderson;Muscle Nerve,1996

4. Deflazacort increases laminin expression and myogenic repair, and induces early persistent functional gain in mdx mouse muscular dystrophy;Anderson;Cell Transplant.,2000

5. Laminin-alpha 2 but not -alpha 1-mediated adhesion of human (Duchenne) and murine (mdx) dystrophic myotubes is seriously defective;Angoli;FEBS Lett.,1997

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