Mitochondrial dysfunction in chromaffin cells from the R6/1 mouse model of Huntington's disease: Impact on exocytosis and calcium current regulation-Reference-Cited by-同舟云学术

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Mitochondrial dysfunction in chromaffin cells from the R6/1 mouse model of Huntington's disease: Impact on exocytosis and calcium current regulation

Published:2023-04 Issue: Volume:179 Page:106046
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Fernández Ana,Martínez-Ramírez Carmen,Gómez Ana,de Diego Antonio M.G.,Gandía Luis,Casarejos María José,García Antonio G.

Publisher

Elsevier BV

Subject

Neurology

Reference42 articles.

1. Impaired glutamate transport and glutamate-glutamine cycling: downstream effects of the Huntington mutation;Behrens;Brain,2002

2. The R6/2 transgenic mouse model of Huntington’s disease develops diabetes due to deficient beta-cell mass and exocytosis;Bjorkqvist;Hum. Mol. Genet.,2005

3. Chromaffin cells of the adrenal medulla: physiology, pharmacology, and disease;Carbone;Compr. Physiol.,2019

4. Investigating the transition of pre-symptomatic to symptomatic Huntington’s disease status based on omics data;Christodoulou;Int. J. Mol. Sci.,2020

5. Systemic manifestation and contribution of peripheral tissues to Huntington’s disease pathogenesis;Chuang;Ageing Res. Rev.,2021

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