Isoform-specific dysregulation of AMP-activated protein kinase signaling in a non-human primate model of Alzheimer's disease-Reference-Cited by-同舟云学术

Isoform-specific dysregulation of AMP-activated protein kinase signaling in a non-human primate model of Alzheimer's disease

Published:2021-10 Issue: Volume:158 Page:105463
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Wang Xin,Zhou Xueyan,Uberseder Beth,Lee Jingyun,Latimer Caitlin S.,Furdui Cristina M.,Keene C. Dirk,Montine Thomas J.,Register Thomas C.,Craft Suzanne,Shively Carol A.,Ma Tao

Funder

National Institutes of Health

Alzheimer's Association

BrightFocus Foundation

Nancy and Buster Alvord Endowment

Publisher

Elsevier BV

Subject

Neurology

Reference43 articles.

1. Alzheimer’s disease facts and figures;Alzheimer’’s Association;Alzheimers Dement.,2020

2. Genetic reduction of eEF2 kinase alleviates pathophysiology in Alzheimer’s disease model mice;Beckelman;J. Clin. Invest.,2019

3. Translational control of long-lasting synaptic plasticity and memory;Costa-Mattioli;Neuron.,2009

4. Successful therapies for Alzheimer’s disease: why so many in animal models and none in humans?;Franco;Front. Pharmacol.,2014

5. Aβ immunotherapy for Alzheimer’s disease: where are we?;Güell-Bosch;Neurodegen. Dis. Manag.,2016

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