ALS-causing SOD1 mutants regulate occludin phosphorylation/ubiquitination and endocytic trafficking via the ITCH/Eps15/Rab5 axis-Reference-Cited by-同舟云学术

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ALS-causing SOD1 mutants regulate occludin phosphorylation/ubiquitination and endocytic trafficking via the ITCH/Eps15/Rab5 axis

Published:2021-06 Issue: Volume:153 Page:105315
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Tang Jingshu,Kang Yuying,Zhou Yujun,Li Xinnan,Lan Jiaqi,Wu Lei,Feng Xinhong,Peng Ying

Funder

National Natural Science Foundation of China

Fundamental Research Funds for the Central Universities

Publisher

Elsevier BV

Subject

Neurology

Reference52 articles.

1. Serum and CSF immunological findings in ALS;Apostolski;Acta Neurol. Scand.,1991

2. Oxidative stress in ALS: a mechanism of neurodegeneration and a therapeutic target;Barber;Biochim. Biophys. Acta,2006

3. The blood-spinal cord barrier: morphology and clinical implications;Bartanusz;Ann. Neurol.,2011

4. Inhibition of clathrin-coated pit assembly by an Eps15 mutant;Benmerah;J. Cell Sci.,1999

5. Amyotrophic lateral sclerosis;Brown;N. Engl. J. Med.,2017

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4. Patching Up the Permeability: The Role of Stem Cells in Lessening Neurovascular Damage in Amyotrophic Lateral Sclerosis;Stem Cells Translational Medicine;2022-10-01

5. Cold-induced asthma exacerbation through disruption of murine airway epithelial barrier;2022-08-08

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