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SGK1.1 limits brain damage after status epilepticus through M current-dependent and independent mechanisms

  • Published:2021-06 Issue: Volume:153 Page:105317
  • ISSN:0969-9961
  • Container-title:Neurobiology of Disease
  • language:en
  • Short-container-title:Neurobiology of Disease

Author:

Martin-Batista Elva,Maglio Laura E.,Armas-Capote Natalia,Hernández Guadalberto,Alvarez de la Rosa Diego,Giraldez Teresa

Funder

Government of Spain Ministry of Science and Innovation

European Research Council

Publisher

Elsevier BV

Subject

Neurology

Reference65 articles.

1. EMD638683 , a novel SGK inhibitor with antihyper- tensive potency;Ackermann;Cell. Physiol. Biochem.,2011

2. The dopamine transporter is differentially regulated after dopaminergic lesion;Afonso-Oramas;Neurobiol. Dis.,2010

3. Vigabatrin protects against hippocampal damage but is not antiepileptogenic in the lithium-pilocarpine model of temporal lobe epilepsy;André;Epilepsy Res.,2001

4. The Serum and Glucocorticoids Regulated Kianse 1.1 (SGK1.1) as an Anticonvulsant Factor;Armas-Capote,2017

5. SGK1.1 reduces Kainic acid-induced seizure severity and leads to rapid termination of seizures;Armas-Capote;Cereb. Cortex,2020

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