Spreading of amyloid-β peptides via neuritic cell-to-cell transfer is dependent on insufficient cellular clearance-Reference-Cited by-同舟云学术

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Spreading of amyloid-β peptides via neuritic cell-to-cell transfer is dependent on insufficient cellular clearance

Published:2014-05 Issue: Volume:65 Page:82-92
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Domert Jakob,Rao Sahana Bhima,Agholme Lotta,Brorsson Ann-Christin,Marcusson Jan,Hallbeck Martin,Nath Sangeeta

Funder

Swedish Alzheimer's Foundation

Hans-Gabriel and Alice Trolle-Wachtmeisters Foundation for Medical Research

Gustav V and Queen Victoria's Foundation

Swedish Dementia Foundation

Linköping University Neurobiology Center

Östergötland County Council

Publisher

Elsevier BV

Subject

Neurology

Reference48 articles.

1. An in vitro model for neuroscience: differentiation of SH-SY5Y cells into cells with morphological and biochemical characteristics of mature neurons;Agholme;J. Alzheimers Dis.,2010

2. Amyloid-beta secretion, generation, and lysosomal sequestration in response to proteasome inhibition: involvement of autophagy;Agholme;J. Alzheimers Dis.,2012

3. Structural conversion of neurotoxic amyloid-beta(1–42) oligomers to fibrils;Ahmed;Nat. Struct. Mol. Biol.,2010

4. Live-cell imaging of autophagy induction and autophagosome–lysosome fusion in primary cultured neurons;Bains;Methods Enzymol.,2009

5. The toxic Abeta oligomer and Alzheimer's disease: an emperor in need of clothes;Benilova;Nat. Neurosci.,2012

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