Glutamate excitotoxicity in neurons triggers mitochondrial and endoplasmic reticulum accumulation of Parkin, and, in the presence of N-acetyl cysteine, mitophagy-Reference-Cited by-同舟云学术

Glutamate excitotoxicity in neurons triggers mitochondrial and endoplasmic reticulum accumulation of Parkin, and, in the presence of N-acetyl cysteine, mitophagy

Published:2015-02 Issue: Volume:74 Page:180-193
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Van Laar Victor S.,Roy Nikita,Liu Annie,Rajprohat Swati,Arnold Beth,Dukes April A.,Holbein Cory D.,Berman Sarah B.

Funder

National Institutes of Health

American Federation for Aging Research

Ellison Medical Foundation/AFAR Postdoctoral Fellowship

Parkinson Disease Foundation

Publisher

Elsevier BV

Subject

Neurology

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3. Mitophagy of damaged mitochondria occurs locally in distal neuronal axons and requires PINK1 and Parkin;Ashrafi;J. Cell Biol.,2014

4. Glutamate transporter currents in bergmann glial cells follow the time course of extrasynaptic glutamate;Bergles;Proc. Natl. Acad. Sci. U. S. A.,1997

5. An update on the potential role of excitotoxicity in the pathogenesis of Parkinson's disease;Blandini;Funct. Neurol.,2010

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