Transgenic mice over-expressing GABABR1a receptors acquire an atypical absence epilepsy-like phenotype-Reference-Cited by-同舟云学术

Transgenic mice over-expressing GABABR1a receptors acquire an atypical absence epilepsy-like phenotype

Published:2007-05 Issue:2 Volume:26 Page:439-451
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Wu Ying,Chan Katherine F.Y.,Eubanks James H.,Guin Ting Wong C.,Cortez Miguel A.,Shen Liqing,Che Liu Chun,Perez Velazquez Jose,Tian Wang Yu,Jia Zhengping,Carter Snead O.

Publisher

Elsevier BV

Subject

Neurology

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5. γ-Hydroxybutyric acid induced spike and wave discharges in rats: relation to high affinity (3H) γ-Hydroxybutyric acid binding sites in the thalamus and cortex;Banerjee;Neuroscience,1993

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