KCa3.1 deficiency attenuates neuroinflammation by regulating an astrocyte phenotype switch involving the PI3K/AKT/GSK3β pathway-Reference-Cited by-同舟云学术

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KCa3.1 deficiency attenuates neuroinflammation by regulating an astrocyte phenotype switch involving the PI3K/AKT/GSK3β pathway

Published:2019-12 Issue: Volume:132 Page:104588
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Wei Tianjiao,Wang Yanxia,Xu Weirong,Liu Yan,Chen Hongzhuan,Yu Zhihua

Funder

National Natural Science Foundation of China

Science and Technology Commission of Shanghai Municipality

Publisher

Elsevier BV

Subject

Neurology

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3. An anti-diabetes agent protects the mouse brain from defective insulin signaling caused by Alzheimer's disease- associated Abeta oligomers;Bomfim;J. Clin. Invest.,2012

4. Short-term high-fat-and-fructose feeding produces insulin signaling alterations accompanied by neurite and synaptic reduction and astroglial activation in the rat hippocampus;Calvo-Ochoa;J. Cereb. Blood Flow Metab.,2014

5. Selective inhibition of KCa3.1 channels mediates adenosine regulation of the motility of human T cells;Chimote;J. Immunol.,2013

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