Fast motor axon loss in SMARD1 does not correspond to morphological and functional alterations of the NMJ-Reference-Cited by-同舟云学术

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Fast motor axon loss in SMARD1 does not correspond to morphological and functional alterations of the NMJ

Published:2013-06 Issue: Volume:54 Page:169-182
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Krieger Frank,Elflein Nicole,Ruiz Rocio,Guerra Joana,Serrano Antonio L.,Asan Esther,Tabares Lucia,Jablonka Sibylle

Funder

Deutsche Forschungsgemeinsschaft

Fundació Marató TV3

FIS-PS09/01267

CIBERNED

Publisher

Elsevier BV

Subject

Neurology

Reference59 articles.

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2. End-plates, transmission and contractile characteristics of muscles without spindles in the hereditary sensory neuropathy of the Sprawling mouse;Brook;Brain,1990

3. Ultrastructural evidence indicating reorganization at the neuromuscular junction in the normal rat soleus muscle;Cardasis;Anat. Rec.,1981

4. Overexpression of growth-associated proteins in the neurons of adult transgenic mice;Caroni;J. Neurosci. Methods,1997

5. Senataxin, the yeast Sen1p orthologue: characterization of a unique protein in which recessive mutations cause ataxia and dominant mutations cause motor neuron disease;Chen;Neurobiol. Dis.,2006

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4. Muscle fiber-type selective propensity to pathology in the nmd mouse model of SMARD1;Biochemical and Biophysical Research Communications;2019-08

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