Chemotherapy promotes astrocytic response to Aβ deposition, but not Aβ levels, in a mouse model of amyloid and APOE-Reference-Cited by-同舟云学术

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Chemotherapy promotes astrocytic response to Aβ deposition, but not Aβ levels, in a mouse model of amyloid and APOE

Published:2022-12 Issue: Volume:175 Page:105915
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Ng Christi Anne S.,Biran Lucas P.,Galvano Elena,Mandelblatt Jeanne,Vicini Stefano,Rebeck G. William

Funder

National Institute on Aging

National Cancer Institute

National Institute of Neurological Disorders and Stroke

National Institutes of Health

Publisher

Elsevier BV

Subject

Neurology

Reference77 articles.

1. Apolipoprotein E epsilon4 is associated with disease-specific effects on brain atrophy in Alzheimer’s disease and frontotemporal dementia;Agosta;Proc. Natl. Acad. Sci. U. S. A.,2009

2. Candidate mechanisms for chemotherapy-induced cognitive changes;Ahles;Nat. Rev. Cancer,2007

3. The relationship of APOE genotype to neuropsychological performance in long-term cancer survivors treated with standard dose chemotherapy;Ahles;Psychooncology.,2003

4. Longitudinal assessment of cognitive changes associated with adjuvant treatment for breast cancer: impact of age and cognitive reserve;Ahles;J. Clin. Oncol.,2010

5. Cancer- and cancer treatment-associated cognitive change: an update on the state of the science;Ahles;J. Clin. Oncol.,2012

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1. Alzheimer disease–related biomarkers and cancer-related cognitive decline: the Thinking and Living with Cancer study;JNCI: Journal of the National Cancer Institute;2024-05-24

2. Neuroimaging based biotypes for precision diagnosis and prognosis in cancer-related cognitive impairment;Frontiers in Medicine;2023-08-29

3. Chemobrain: An accelerated aging process linking adenosine A2A receptor signaling in cancer survivors;International Review of Neurobiology;2023

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