Senescence may mediate conversion of tau phosphorylation-induced apoptotic escape to neurodegeneration-Reference-Cited by-同舟云学术

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Senescence may mediate conversion of tau phosphorylation-induced apoptotic escape to neurodegeneration

Published:2015-08 Issue: Volume:68 Page:82-86
ISSN:0531-5565
Container-title:Experimental Gerontology
language:en
Short-container-title:Experimental Gerontology

Author:

Wang Jian-Zhi,Wang Zhi-Hao

Funder

Natural Science Foundation of China

Ministry of Science and Technology of China

Alzheimer Association in the United States

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Genetics,Molecular Biology,Aging,Biochemistry

Reference49 articles.

1. Abundant tau filaments and nonapoptotic neurodegeneration in transgenic mice expressing human P301S tau protein;Allen;J. Neurosci.,2002

2. Reversible paired helical filament-like phosphorylation of tau is an adaptive process associated with neuronal plasticity in hibernating animals;Arendt;J. Neurosci.,2003

3. Betaine attenuates Alzheimer-like pathological changes and memory deficits induced by homocysteine;Chai;J. Neurochem.,2013

4. Nmnat2 attenuates tau phosphorylation through activation of PP2A;Cheng;J. Alzheimers Dis.,2013

5. Phosphorylation of tau by death-associated protein kinase 1 antagonizes the kinase-induced cell apoptosis;Duan;J. Alzheimers Dis.,2013

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2. An aging, pathology burden, and glial senescence build-up hypothesis for late onset Alzheimer’s disease;Nature Communications;2023-03-25

3. Current understanding of metal ions in the pathogenesis of Alzheimer’s disease;Translational Neurodegeneration;2020-04-03

4. Ginsenoside Re Inhibits ROS/ASK-1 Dependent Mitochondrial Apoptosis Pathway and Activation of Nrf2-Antioxidant Response in Beta-Amyloid-Challenged SH-SY5Y Cells;Molecules;2019-07-24

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