Cullin-3 intervenes in muscle atrophy in the elderly by mediating the degradation of nAchRs ubiquitination
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Endocrinology,Genetics,Molecular Biology,Aging,Biochemistry
Reference35 articles.
1. The ubiquitin proteasome system in neuromuscular disorders: moving beyond movement;Bachiller;Int. J. Mol. Sci.,2020
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3. Cullin E3 ligase activity is required for myoblast differentiation;Blondelle;J. Mol. Biol.,2017
4. Cullin-3–dependent deregulation of ACTN1 represents a pathogenic mechanism in nemaline myopathy;Blondelle;JCI Insight,2019
5. Cullin-3 dependent deregulation of ACTN1 represents a new pathogenic mechanism in nemaline myopathy;Blondelle;JCI Insight,2019
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