Systems modelling predicts chronic inflammation and genomic instability prevent effective mitochondrial regulation during biological ageing-Reference-Cited by-同舟云学术

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Systems modelling predicts chronic inflammation and genomic instability prevent effective mitochondrial regulation during biological ageing

Published:2022-09 Issue: Volume:166 Page:111889
ISSN:0531-5565
Container-title:Experimental Gerontology
language:en
Short-container-title:Experimental Gerontology

Author:

Guimera Alvaro Martinez,Clark Peter,Wordsworth James,Anugula Sharath,Rasmussen Lene Juel,Shanley Daryl P.

Funder

NC3Rs

Novo Nordisk Fonden

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Genetics,Molecular Biology,Aging,Biochemistry

Reference47 articles.

1. Age-related loss of stress-induced nuclear proteasome activation is due to low PARP-1 activity;Bakondi;Free Radic. Biol. Med.,2011

2. Life span extension and neuronal cell protection by drosophila nicotinamidase;Balan;J. Biol. Chem.,2008

3. Nicotinamide riboside promotes Sir2 silencing and extends lifespan via Nrk and Urh1/Pnp1/Meu1 pathways to NAD+;Belenky;Cell,2007

4. CD38 dictates age-related NAD decline and mitochondrial dysfunction through an SIRT3-dependent mechanism;Camacho-Pereira;Cell Metab.,2016

5. AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activity;Canto;Nature,2009

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4. Unlocking Mitochondrial Dysfunction-Associated Senescence (MiDAS) with NAD⁺– a Boolean Model of Mitochondrial Dynamics and Cell Cycle Control;2023-12-18

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