Herpes simplex virus co-infection-induced Chlamydia trachomatis persistence is not mediated by any known persistence inducer or anti-chlamydial pathway-Reference-Cited by-同舟云学术

Herpes simplex virus co-infection-induced Chlamydia trachomatis persistence is not mediated by any known persistence inducer or anti-chlamydial pathway

Published:2008-03-01 Issue:3 Volume:154 Page:971-978
ISSN:1350-0872
Container-title:Microbiology
language:en
Short-container-title:

Author:

Vanover Jennifer¹,Sun Jingru¹,Deka Srilekha²,Kintner Jennifer¹,Duffourc Michelle M.³,Schoborg Robert V.¹

Affiliation:

1. Department of Microbiology, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN 37614-0579, USA

2. ViroMed Laboratories, Minnetonka, MN, USA

3. Department of Pharmacology, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN, USA

Publisher

Microbiology Society

Subject

Microbiology

Reference43 articles.

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2. TLR2-mediated proinflammatory cytokine and chemokine production by microglial cells in response to herpes simplex virus;Aravalli;J Immunol,2005

3. Tryptophan depletion as a mechanism of gamma interferon-mediated chlamydial persistence;Beatty;Infect Immun,1994

4. Transcriptome analysis of chlamydial growth during IFN-gamma-mediated persistence and reactivation;Belland;Proc Natl Acad Sci U S A,2003

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