Emergence of infectious simian virus 40 whose AT tract in the replication origin/early promoter region is substituted by cellular or viral DNAs
Author:
Affiliation:
1. Institute of Molecular Life Sciences, University of Zürich, Winterthurer Str. 190, CH-8057 Zürich, Switzerland
2. Transplantation & Clinical Virology, Dept. of Biomedicine, University of Basel, CH-4003 Basel, Switzerland
Publisher
Microbiology Society
Subject
Virology
Reference39 articles.
1. Large T Antigens of Polyomaviruses: Amazing Molecular Machines
2. Expression of a β-globin gene is enhanced by remote SV40 DNA sequences
3. All six GC-motifs of the SV40 early upstream element contribute to promoter activity in vivo and in vitro.;Barrera-Saldana;EMBO J,1985
4. Deletions covering the putative promoter region of early mRNAs of simian virus 40 do not abolish T-antigen expression.
5. Territorial limits and functional anatomy of the simian virus 40 replication origin.
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1. Novel Human Polyomavirus Noncoding Control Regions Differ in Bidirectional Gene Expression according to Host Cell, Large T-Antigen Expression, and Clinically Occurring Rearrangements;Journal of Virology;2018-04
2. Sp1 Sites in the Noncoding Control Region of BK Polyomavirus Are Key Regulators of Bidirectional Viral Early and Late Gene Expression;Journal of Virology;2015-03-15
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