The cellular endosomal protein stannin inhibits intracellular trafficking of human papillomavirus during virus entry-Reference-Cited by-同舟云学术

The cellular endosomal protein stannin inhibits intracellular trafficking of human papillomavirus during virus entry

Published:2017-11-01 Issue:11 Volume:98 Page:2821-2836
ISSN:0022-1317
Container-title:Journal of General Virology
language:en
Short-container-title:

Author:

Lipovsky Alex¹,Erden Asu¹,Kanaya Eriko¹,Zhang Wei²,Crite Mac³,Bradfield Clinton⁴,MacMicking John⁴⁵,DiMaio Daniel²⁶,Schoggins John W.⁷,Iwasaki Akiko¹⁵⁶

Affiliation:

1. Department of Immunobiology, Yale School of Medicine, New Haven, CT 06520-8011, P.O. Box 208011, USA

2. Department of Genetics, Yale School of Medicine, New Haven, CT 06520-8005, P.O. Box 208005, USA

3. Microbiology Graduate Program, Yale School of Medicine, New Haven, CT 06519, 295 Congress Avenue, USA

4. Department of Microbial Pathogenesis, Yale School of Medicine, New Haven, CT 06536-0812, P.O. Box 9812, USA

5. Howard Hughes Medical Institute, Chevy Chase, MD 20814, USA

6. Yale Cancer Center, New Haven, CT 06520-8028, PO Box 208028, USA

7. Department of Microbiology, University of Texas Southwestern Medical Center, 5323 Harry hines Blvd., Dallas, TX 75390, USA

Publisher

Microbiology Society

Subject

Virology

Reference62 articles.

1. The global health burden of infection-associated cancers in the year 2002

2. Human papillomavirus in cervical and head-and-neck cancer

3. Multiple Heparan Sulfate Binding Site Engagements Are Required for the Infectious Entry of Human Papillomavirus Type 16

4. Kallikrein-8 Proteolytically Processes Human Papillomaviruses in the Extracellular Space To Facilitate Entry into Host Cells

5. Cleavage of the papillomavirus minor capsid protein, L2, at a furin consensus site is necessary for infection

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