Divergent chemokine, cytokine and β-defensin responses to gastric candidiasis in immunocompetent C57BL/6 and BALB/c mice-Reference-Cited by-同舟云学术

Divergent chemokine, cytokine and β-defensin responses to gastric candidiasis in immunocompetent C57BL/6 and BALB/c mice

Published:2005-01-01 Issue:1 Volume:54 Page:87-92
ISSN:0022-2615
Container-title:Journal of Medical Microbiology
language:en
Short-container-title:

Author:

Schofield David A¹,Westwater Caroline¹,Balish Edward¹

Affiliation:

1. Department of Microbiology and Immunology1 and Department of Stomatology2, Medical University of South Carolina, 173 Ashley Avenue, Charleston, SC 29403, USA

Publisher

Microbiology Society

Subject

Microbiology (medical),General Medicine,Microbiology

Reference16 articles.

1. Murine candidiasis: susceptibility is associated with the induction of T cell-mediated, strain-specific autoreactivity;Ashman;Immunol Cell Biol,1990

2. Murine candidiasis.Pathogenesis and host responses in genetically distinct inbred mice;Ashman;Immunol Cell Biol,1987

3. Candidiasis in interferon-gamma knockout (IFN-gamma-/-) mice;Balish;J Infect Dis,1998

4. Mucosal and systemic candidiasis in IL-8Rh-/- BALB/c mice;Balish;J Leukoc Biol,1999

5. Murine β-defensin-3 is an inducible peptide with limited tissue expression and broad-spectrum antimicrobial activity;Burd;Shock,2002

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Gut mycobiome: A “black box” of gut microbiome–host interactions;WIREs Mechanisms of Disease;2023-05-08

2. Absence of Bacteria Permits Fungal Gut-To-Brain Translocation and Invasion in Germfree Mice but Ageing Alone Does Not Drive Pathobiont Expansion in Conventionally Raised Mice;Frontiers in Aging Neuroscience;2022-07-18

3. Candida albicans Modulates Murine and Human Beta Defensin-1 during Vaginitis;Journal of Fungi;2021-12-28

4. Gut Microbial and Metabolic Responses to Salmonella enterica Serovar Typhimurium and Candida albicans;mBio;2018-12-21

5. Candida albicans Commensalism and Human Diseases;Mechanisms Underlying Host-Microbiome Interactions in Pathophysiology of Human Diseases;2018