Bacterial interference with canonical NFκB signalling
Author:
Affiliation:
1. Research Group of Host–Microbe Interactions, Department of Medical Biology, Faculty of Health Sciences, University of Tromsø, Tromsø, Norway
Publisher
Microbiology Society
Subject
Microbiology
Reference104 articles.
1. Genetic Alteration of Capsule Type but Not PspA Type Affects Accessibility of Surface-Bound Complement and Surface Antigens of Streptococcus pneumoniae
2. Myeloid differentiation factor 88-dependent signalling controls bacterial growth during colonization and systemic pneumococcal disease in mice
3. An injected bacterial effector targets chromatin access for transcription factor NF-κB to alter transcription of host genes involved in immune responses
4. A bacterial E3 ubiquitin ligase IpaH9.8 targets NEMO/IKKγ to dampen the host NF-κB-mediated inflammatory response
5. Evasion of Toll-like receptor 2 activation by staphylococcal superantigen-like protein 3
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