Ischemic brain damage and mitochodrial dysfunction
Author:
Affiliation:
1. Department of Anesthesiology, Tokyo Medical University
Publisher
The Japanese Society of Cerebral Blood Flow and Metabolism
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/cbfm/26/2/26_155/_pdf
Reference14 articles.
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2. 2) Nolan JP, Neumar RW, Adrie C, Aibiki M, Berg RA, Bbttiger BW, Callaway C, Clark RS, Geocadin RG, Jauch EC, Kern KB, Laurent I, Longstreth WT, Merchant RM, Morley P, Morrison LJ, Nadkarni V, Peberdy MA, Rivers EP, Rodriguez-Nunez A, Sellke FW, Spaulding C, Sunde K, Hoek TV: Post-cardiac arrest syndrome: epidemiology, pathophysiology, treatment, and prognostication: a scientific statement from the International Liaison Committee on Resuscitation; the American Heart Association Emergency Cardiovascular Care Committee; the Council on Cardiovascular Surgery and Anesthesia; the Council on Cardiopulmonary, Perioperative, and Critical Care; the Council on Clinical Cardiology; the Council on Stroke (Part 1). Int Emerg Nurs 17: 203–225, 2009
3. 4) SOS-KANTO study group: Cardiopulmonary resuscitation by bystanders with chest compression only (SOS-KANTO): an observational study. Lancet 369: 920–926, 2007
4. 5) Smith ML, Bendek G, Dahlgren N, Rosén I, Wieloch T, Siesjö BK: Models for studying long-term recovery following forebrain ischemia in the rat. 2. A 2-vessel occlusion model. Acta Neurol Scand 69: 385–401, 1984
5. 6) Cooper HK, Zalewska T, Kawakami S, Hossmann KA, Kleihues P: The effect of ischaemia and recirculation on protein synthesis in the rat brain. J Neurochem 28: 929–934, 1977
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